Ipriflavone Study Table 1

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Table 1. Osteoporosis therapies, mechanisms, benefits ad risks
Medication	Primary Mechanism	Benefits	Risks
Conventional ERT	Inhibits bone resorption	Improves vasomotor symptoms of menopause	Increased risk of estrogen-related cancer, venous thrombosis, hypertension, gallbladder disease.
Calcitonin	Inhibits bone resorption	No estrogenic effect; analgesic effect on bone pain	Secondary hyperparathyroidism; anti-CT antibody production Injectable: nausea, vomiting, vertigo. Intranasal: rhinitis, nasal irritation
Bisphosphonates	Inhibits bone resorption	No estrogenic effect	Gastric erosion, thyroid adenoma (rats); Safety of Tx. > 4 years not known
Sodium Fluoride	Stimulates osteoblasts (bone formation)	Increases bone mineral density for up to 5 years	Abnormal bone crystallinity; no decrease in hip fracture rate; decrease in cortical bone in favor of trabecular bone; acute GI nausea, vomiting, and hemorrhage
Selective Estrogen Receptor Modulators (Raloxifene)	Inhibits bone resorption	Estrogen effect on bone and lipids; not on breast or uterus	Increased risk of venous thrombosis, pulmonary embolism; fatal liver toxicity; ovarian tumors and hepatic cancer (animals)
Ipriflavone	Inhibits osteoclast and enhances osteoblast activity	No estrogen effect; fewer side effects; analgesic effect on bone pain	GI upset

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This information is for general education purposes only and has not been evaluated by the FDA or FTC. The information contained herein is not intended to treat, cure or diagnose any disease. If you suspect you have a health condition of any kind, contact a healthcare professional. Individual results may vary.